Regarding COVID-19 and hydroxychloroquine (HCQ), HCQ is known to increase pH in lysosomes by 1-2 orders of magnitude, and also results in lethal intracellular heme overload in parasites responsible for hemolytic diseases. Question is, if circulating levels of xanthine oxidase (XO) are also known to be increased in hemolytic diseases, if one considers the natural flavonoids Kaempferol, quercetin, or luteolin, which are known xanthine oxidase (XO) inhibitors, antivirals, and anti-oxidants, what would be different about pharmacology of natural XO inhibitors and HCQ?
Is HCQ an XO inhibitor because it results in heme overload in parasites? What about anti-oxidant properties of HCQ in terms of radical scavenging? There is no information supporting the notion that HCQ is an antiviral.
Sticking with hemolytic diseases, for which the goal is overload of intracellular heme in parasites, what would happen if a human took high levels of an XO inhibitor that wasn't HCQ during malaria?
XO inhibitors usually result in an overall decrease in uric acid, essentially rendering them as a natural supplement for gout -- i.e., replacement to allopurinol, which has horrendous side effects.